If we ask an individual what his definition of Hyperuricemia is, maybe he will say that it is when the uric acid in the body is too high. Well, at some point that can be correct and true. Hyperuricemia is when there is an excess amount of uric acid in the blood. That is the standard definition of Hyperuricemia. But, what does it mean when we say that it is a standard definition? We say that it is the standard definition because that is how they usually define it. Or it is a standard definition because that is how they commonly define it. But, the standard definition is not always correct. Do you know that the standard treatment for gout is ineffective? Allopurinol can only pause the production uric acid and it can suddenly rise and cause damage.
There is nothing standard about Hyperuricemia or Gout. The standard treatments for these diseases are unsuccessful. In addition, the standard medications for these diseases can only cause different side effects for the body.
When you read this article, it can help you understand the definition of Hyperuricemia in a not so standard approach.
Hyperuricemia in a different point-of-view
Although the standard definition of Hyperuricemia is correct, it is very limited. Its standard definition addresses just one of the many parts of Hyperuricemia.
We can define Hyperuricemia as “Hyperuricemia is a condition characterized by abnormally high uric acid level in the body, and it has risks to cause diabetes, hypertension, cancer and many other diseases”.
You might think that, is it really possible that high level of uric acid can cause high blood sugar, hypertension, and cancer? Is it really that harmful to the body? Wake up, people, it is.
Hyperuricemia and the huge number of Free Radicals
High levels of uric acid in the body can also mean that there are a huge group of free radicals. A radical is an atom with no pair electron. Electrons need a pair and they will seek a pair until they find it. An unpaired electron can also steal electrons from other atoms. They can also share an unpaired electron. In this way, they can create a pair of an electron with another atom with an unpaired electron. When this happens, the behavior of the atom can also change to gain an electron. Or their behavior changes when they lose their pair of electron.
We can use ourselves as an easier example to understand it better. We humans need a pair. And when we badly need a pair we can also steal each other’s pair. When this happens, it can affect the pair of the pair that we stole. There will be a change of roles with the previous and the present pairs. And the previous pair will not be able to function properly.
When a patient is suffering from Hyperuricemia, it means that he has high levels of uric acid, yes. This can also mean that the patient has groups of Hydrogen atoms. Hydrogen atoms have a single electron. And this electron will find a pair and steal it from another electron from another atom. As the electrons find their pairs, the structure of the atoms of the cells also changes. The electron will become a free radical. And the functions of the cells are also damaged. Because of this, the cells in the body can also be damaged. When that happens, it can cause diabetes.
There many effects of too many acids in the body. There are also many effects of free radicals in the body. And you can read more about how the free radicals can cause hypertension or cancer.
It is important to identify the possible causes and to evaluate the renal function. The non-pharmacological cure is best one to take into contemplation with the patients of hyperuricemia. Hyperuricemia can be considered a part of metabolic syndrome. Hyperuricemia is a risk factor that’s independent cardiovascular and renal mortality rates. But, clinical practices still need to try and finalize. The studies can lead to some new therapeutic approach to hyperuricemia.
If the extra-articular assistances of the hyperuricemia treatment can be confirmed, the hypouricemic therapy can reduce the cardiovascular danger of cured patients. In addition, it can also improve their own life of the patient thru a straight decrement in uric acid-related complications and clinical events.